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dc.creatorMedina-Echeverz, J. (José)-
dc.creatorBerraondo, P. (Pedro)-
dc.date.accessioned2012-09-25T10:34:00Z-
dc.date.available2012-09-25T10:34:00Z-
dc.date.issued2012-
dc.identifier.citationMedina-Echeverz J, Berraondo P. Colon cancer eradication after chemoimmunotherapy is associated with intratumoral emergence of proinflammatory myeloid cells. Oncoimmunology 2012 Jan 1;1(1):118-120.es_ES
dc.identifier.issn2162-402X-
dc.identifier.urihttp://hdl.handle.net/10171/23176-
dc.description.abstractInterleukin-12 immune stimulation lacks efficacy in established solid tumor models. Disruption of tumor microenvironment homeostasis by low-dose cyclophosphamide prior to interleukin-12 gene therapy led to CD8+ T cell-driven established tumor rejection. This only takes place when inflammatory myeloid cells infiltrate the tumor bed, and is crucial for the latter antitumor response.es_ES
dc.language.isoenges_ES
dc.publisherLandes Biosciencees_ES
dc.rightsinfo:eu-repo/semantics/openAccesses_ES
dc.subjectInterleukin-12es_ES
dc.subjectCyclophosphamidees_ES
dc.subjectColorectal canceres_ES
dc.subjectInflammatory myeloid cellses_ES
dc.titleColon cancer eradication after chemoimmunotherapy is associated with intratumoral emergence of proinflammatory myeloid cellses_ES
dc.typeinfo:eu-repo/semantics/articlees_ES
dc.relation.publisherversionhttp://www.ncbi.nlm.nih.gov/pmc/articles/PMC3376964/es_ES
dc.type.driverinfo:eu-repo/semantics/articlees_ES

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