Full metadata record
DC FieldValueLanguage
dc.creatorGarcia-Manero, M. (Manuel)-
dc.creatorOlartecoechea, B. (Begoña)-
dc.creatorRoyo, P. (Pedro)-
dc.creatorAuba, M. (M.)-
dc.creatorLópez-García, G. (Guillermo)-
dc.date.accessioned2014-03-07T09:12:39Z-
dc.date.available2014-03-07T09:12:39Z-
dc.date.issued2008-
dc.identifier.citationGarcía Manero M, Olartecoechea B, Royo Manero P, Aubá M, López G. Endometriosis. Rev Med Univ Navarra. 2009 Apr-Jun;53(2):4-7.es_ES
dc.identifier.issn0556-6177-
dc.identifier.urihttp://hdl.handle.net/10171/35393-
dc.description.abstractEndometriosis is a common gynaecological disease of unknown aetiology which affects an estimated 10% to 15% of all premenopausal women. It is defined as the presence of endometrial tissue, consisting of both glandular epithelium and stroma, outside the uterine cavity. Three different clinical entities of endometriosis can be distinguished: peritoneal endometriosis, ovarian endometriosis and deep invasive endometriosis. There are several theories to explain their pathogenesis: metaplasia of the mesothelium, in situ development of Müllerian remnants in the rectovaginal area (deep-invasive lesions) or retrograde transplantation of shed menstrual effluent (peritoneal implants). The most widely accepted hypothesis for the development of endometriosis is retrograde menstruation. However, some other factor renders certain women susceptible to the implantation and growth of this ectopic endometriumes_ES
dc.language.isospaes_ES
dc.publisherEdiciones Universidad de Navarraes_ES
dc.rightsinfo:eu-repo/semantics/openAccesses_ES
dc.rightsinfo:eu-repo/semantics/openAccesses_ES
dc.subjectEndometriosises_ES
dc.subjectEpidemiologíaes_ES
dc.subjectEtiopatogeniaes_ES
dc.titleEndometriosises_ES
dc.typeinfo:eu-repo/semantics/articlees_ES

Files in This Item:
File: 
Rev Med Univ Navarra 2008 endometriosis[1].pdf
Description: 
Size: 
31,53 kB
Format: 
Adobe PDF


Items in Dadun are protected by copyright, with all rights reserved, unless otherwise indicated.